Pages listed in BOTH Dental and Infants
- Half as many tooth enamel defects in child if 2400 IU Vitamin D in late pregnancy – RCT Aug 2019
- Half as many teeth problems if higher cord blood level of vitamin D – March 2019
- No tooth decay in children with adequate Vitamin D (small study) – June 2018
- Early tooth decay 1.9 X more likely if a poor Vitamin D receptor – July 2017
- Severe childhood dental problems 2.4 X more likely if breastfed for more than two years (low vitamin D) – June 2017
- Dental Caries risk in children cut in half if more than 20 nanogram of Vitamin D – Nov 2015
- Teeth enamel problems (MIH) 11 percent less likely with just 4 ng more vitamin D – Dec 2014
- 3X fewer infant dental caries if good level of vitamin D while pregnant – April 2014
- Severe tooth decay in children unless supplemented with Vitamin D drops – Oct 2013
- UVB added in classroom reduced cavities, increased height, increased academics. etc
- Dental caries cut in half by vitamin D, review of 24 old clinical trials – Nov 2012
- Children with severe caries were 2X more likely to have inadequate vitamin D – July 2012
Download the PDF (from Scribner) from VitaminDWiki
Robert J. Schroth, DMD, MSc, PhDa,b,c, Christopher Lavelle, DDS, PhD, DSc, MBA, FRCPathd, Robert Tate, PhDe, Sharon Bruce, BN, MA, PhDe, Ronald J. Billings, DDS, MSDf, and Michael E.K. Moffatt, MD, MSc, FRCPCc,e
A Preventive Dental Science, Faculty of Dentistry,
B Pediatrics and Child Health, Faculty of Medicine,
D Oral Biology, Faculty of Dentistry,
E Community Health Sciences, Faculty of Medicine, University of Manitoba, Winnipeg, Manitoba, Canada;
C The Manitoba Institute of Child Health, Winnipeg, Manitoba, Canada;
F Division of Community Dentistry and Oral Disease Prevention, Eastman Institute for Oral
OBJECTIVES: Inadequate maternal vitamin D (assessed by using 25-hydroxyvitamin D [25OHD]) levels during pregnancy may affect tooth calcification, predisposing enamel hypoplasia and early childhood caries (ECC). The purpose of this study was to determine the relationship between prenatal 25OHD concentrations and dental caries among offspring during the first year of life.
METHODS: This prospective cohort study recruited expectant mothers from an economically disadvantaged urban area. A prenatal questionnaire was completed and serum sample drawn for 25OHD. Dental examinations were completed at 1 year of age while the parent/caregiver completed a questionnaire. The examiner was blinded to mothers’ 25OHD levels. A P value ≤ .05 was considered significant.
RESULTS: Overall, 207 women were enrolled (mean age: 19 ± 5 years). The mean 25OHD level was 48 ± 24 nmol/L, and 33% had deficient levels. Enamel hypoplasia was identified in 22% of infants; 23% had cavitated ECC, and 36% had ECC when white spot lesions were included in the assessment.
Mothers of children with ECC had significantly lower 25OHD levels than those whose children were caries-free (41 ± 20 vs 52 ± 27 nmol/L; P = .05). Univariate Poisson regression analysis for the amount of untreated decay revealed an inverse relationship with maternal 25OHD.
Logistic regression revealed that
- enamel hypoplasia (P < .001),
- infant age (P = .002), and
- lower prenatal 25OHD levels (P = .02)
were significantly associated with ECC.
CONCLUSIONS: This study found that maternal prenatal 25OHD levels may have an influence on the primary dentition and the development of ECC.Title was revised Aug 2019 caused the visitor count to reset.
There have actually been